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  4. Immunopathological signatures in multisystem inflammatory syndrome in children and pediatric COVID-19
Details

Immunopathological signatures in multisystem inflammatory syndrome in children and pediatric COVID-19

Journal
Nature Medicine
ISSN
1078-8956
1546-170X
Date Issued
2022
Author(s)
Keith Sacco
Riccardo Castagnoli
Svetlana Vakkilainen
Can Liu
Ottavia M. Delmonte
Cihan Oguz
Ian M. Kaplan
Sara Alehashemi
Peter D. Burbelo
Farzana Bhuyan
Adriana A. de Jesus
Kerry Dobbs
Lindsey B. Rosen
Aristine Cheng
Elana Shaw
Mikko S. Vakkilainen
Francesca Pala
Justin Lack
Yu Zhang
Danielle L. Fink
Vasileios Oikonomou
Andrew L. Snow
Clifton L. Dalgard
Jinguo Chen
Brian A. Sellers
Gina A. Montealegre Sanchez
Karyl Barron
REY JURADO, EMMA  
Facultad de Medicina Clínica Alemana Universidad del Desarrollo  
VIAL COX, MARIA CECILIA  
Facultad de Medicina Clínica Alemana Universidad del Desarrollo  
POLI HARLOWE, MARIA CECILIA BERTA  
Facultad de Medicina Clínica Alemana Universidad del Desarrollo  
Amelia Licari
Daniela Montagna
Gian Luigi Marseglia
Francesco Licciardi
Ugo Ramenghi
Valentina Discepolo
Andrea Lo Vecchio
Alfredo Guarino
Eli M. Eisenstein
Luisa Imberti
Alessandra Sottini
Andrea Biondi
Sayonara Mató
Dana Gerstbacher
Meng Truong
Michael A. Stack
Mary Magliocco
Marita Bosticardo
Tomoki Kawai
Jeffrey J. Danielson
Tyler Hulett
Manor Askenazi
Shaohui Hu
Jason Barnett
Xi Cheng
Krishnaveni Kaladi
Vasudev Kuram
Joseph Mackey
Neha M. Bansal
Andrew J. Martins
Boaz Palterer
Helen Matthews
Uma Mudunuri
Marshall Nambiar
Andrew J. Oler
Andre Rastegar
Smilee Samuel
Conrad Shyu
Varsha Waingankar
Sarah Weber
Sandhya Xirasagar
Yazmin Espinosa
Camila Astudillo
Cecilia Piñera
Ricardo González
Maria De Filippo
Martina Votto
Lorenza Montagna
Jeffrey I. Cohen
Helen C. Su
Douglas B. Kuhns
Michail S. Lionakis
Thomas M. Snyder
Steven M. Holland
Raphaela Goldbach-Mansky
John S. Tsang
Luigi D. Notarangelo
Type
Resource Types::text::journal::journal article
Scopus ID
2-s2.0-85126232677
WoS ID
WOS:000757243200002
DOI
10.1038/s41591-022-01724-3
URL
https://investigadores.udd.cl/handle/123456789/5659
URL Institutional Repository
http://hdl.handle.net/11447/5952
Cite this document
Sacco, K., Castagnoli, R., Vakkilainen, S., Liu, C., Delmonte, O. M., Oguz, C., Kaplan, I. M., Alehashemi, S., Burbelo, P. D., Bhuyan, F., De Jesus, A. A., Dobbs, K., Rosen, L. B., Cheng, A., Shaw, E., Vakkilainen, M. S., Pala, F., Lack, J., Zhang, Y., … Notarangelo, L. D. (2022). Immunopathological signatures in multisystem inflammatory syndrome in children and pediatric COVID-19. Nature Medicine, 28(5), 1050-1062. https://doi.org/10.1038/s41591-022-01724-3
Project(s)
Mecanismo Inmunologicos Protectores y Biomarcadores de Respuesta Inmmune en Covid 19 Identificación de Dianas Terapeuticas Para la Recomendación de Terapias Inmunomoduladoras de Uso Precoz  
Unraveling the differences between mild and severe hantavirus infected patients: delineating mechanisms to propose novel therapies  
Deciphering the mechanisms of disease and therapeutic targets for proteasome maturation protein (pomp) related autoinflammation and immune dysregulation  
Dataset(s)
Dataset - Multiomics approach identifies novel age-, time- and treatment-related immunopathological signatures in MIS-C and pediatric COVID-19  
Dataset - Immunopathological signatures in multisystem inflammatory syndrome in children and pediatric COVID-19  
Subjects
child

; 

covid-19

; 

humans

; 

sars-cov-2

; 

systemic inflammatory response syndrome

; 

t-lymphocytes

; 

anakinra

; 

biological product

; 

gamma interferon

; 

glucocorticoid

; 

hla a2 antigen

; 

hla b antigen

; 

hla c antigen

; 

immunoglobulin

; 

infliximab

; 

inotropic agent

; 

interferon

; 

tocilizumab

; 

virus spike protein

; 

allele

; 

article

; 

cell expansion

; 

child

; 

controlled study

; 

female

; 

gene expression

; 

genetic susceptibility

; 

human

; 

human cell

; 

immunopathology

; 

longitudinal study

; 

major clinical study

; 

male

; 

multiomics

; 

pediatric multisystem inflammatory syndrome

; 

protein blood level

; 

proteomics

; 

single cell analysis

; 

t lymphocyte

; 

t lymphocyte activation

; 

complication

; 

genetics

; 

systemic inflammatory response syndrome
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