Interferon-γ and high glucose-induced opening of Cx43 hemichannels causes endothelial cell dysfunction and damage
Journal
Biochimica et Biophysica Acta (BBA) - Molecular Cell Research
ISSN
0167-4889
Date Issued
2020
Author(s)
Juan C. Sáez
Susana Contreras-Duarte
Valeria C. Labra
Cristian A. Santibañez
Luis A. Mellado
Carla A. Inostroza
Tanhia F. Alvear
Victoria Velarde
Juan A. Orellana
Type
Resource Types::text::journal::journal article
URL Institutional Repository
Cite this document
Sáez, J. C., Contreras-Duarte, S., Labra, V. C., Santibañez, C. A., Mellado, L. A., Inostroza, C. A., Alvear, T. F., Retamal, M. A., Velarde, V., & Orellana, J. A. (2020). Interferon-γ and high glucose-induced opening of Cx43 hemichannels causes endothelial cell dysfunction and damage. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research, 1867(8), 118720. https://doi.org/10.1016/j.bbamcr.2020.118720
Subjects
connexons
;
diabetes
;
endothelium
;
hemichannels
;
inflammation
;
cell death
;
cell line
;
connexin 43
;
diabetes mellitus
;
dinoprostone
;
endothelial cells
;
endothelium
;
glucose
;
humans
;
inflammation
;
insulin
;
interferon-gamma
;
nitric oxide
;
signal transduction
;
superoxides
;
adenosine triphosphate
;
calcium ion
;
connexin 43
;
cyclooxygenase 2
;
gamma interferon
;
glucose
;
inducible nitric oxide synthase
;
interleukin 1beta
;
mitogen activated protein kinase 14
;
nitric oxide
;
prostaglandin e1
;
prostaglandin e2
;
purinergic p2x7 receptor
;
purinergic p2y1 receptor
;
superoxide
;
tumor necrosis factor
;
connexin 43
;
gamma interferon
;
gja1 protein, human
;
glucose
;
insulin
;
nitric oxide
;
prostaglandin e2
;
superoxide
;
article
;
calcium cell level
;
cell communication
;
cell damage
;
cell death
;
cell survival
;
controlled study
;
cytoplasm
;
endothelial dysfunction
;
endothelium cell
;
gap junction
;
glucose transport
;
human
;
human cell
;
insulin signaling
;
live cell imaging
;
priority journal
;
protein function
;
signal transduction
;
cell line
;
cytology
;
diabetes mellitus
;
drug effect
;
endothelium
;
endothelium cell
;
inflammation
;
metabolism