GAJARDO VIDAL, ANDREA ELIZABETH

Preferred name

Main Affiliation

Facultad de Gobierno

agajardo@udd.cl

ORCID

0000-0003-3946-0795

Scopus Author ID

57194767907

Goggle Scholar

https://scholar.google.com/citations?user=xzJ_kvgAAAAJ&hl=en

Now showing 1 - 10 of 21

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How right hemisphere damage after stroke can impair speech comprehension
(2018)
GAJARDO VIDAL, ANDREA ELIZABETH
;
Diego L Lorca-Puls
;
Thomas M H Hope
;
Oiwi Parker Jones
;
Mohamed L Seghier
4Scopus© Citations 67
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Degeneracy in the neurological model of auditory speech repetition
(2023)
Noor Sajid
;
GAJARDO VIDAL, ANDREA ELIZABETH
;
Justyna O. Ekert
;
Diego L. Lorca-Puls
;
Thomas M. H. Hope
<jats:title>Abstract</jats:title><jats:p>Both classic and contemporary models of auditory word repetition involve at least four left hemisphere regions: primary auditory cortex for processing sounds; pSTS (within Wernicke’s area) for processing auditory images of speech; pOp (within Broca’s area) for processing motor images of speech; and primary motor cortex for overt speech articulation. Previous functional-MRI (fMRI) studies confirm that auditory repetition activates these regions, in addition to many others. Crucially, however, contemporary models do not specify how regions interact and drive each other during auditory repetition. Here, we used dynamic causal modelling, to test the functional interplay among the four core brain regions during single auditory word and pseudoword repetition. Our analysis is grounded in the principle of degeneracy—i.e., many-to-one structure-function relationships—where multiple neural pathways can execute the same function. Contrary to expectation, we found that, for both word and pseudoword repetition, (i) the effective connectivity between pSTS and pOp was predominantly bidirectional and inhibitory; (ii) activity in the motor cortex could be driven by either pSTS or pOp; and (iii) the latter varied both within and between individuals. These results suggest that different neural pathways can support auditory speech repetition. This degeneracy may explain resilience to functional loss after brain damage.</jats:p>
2Scopus© Citations 3
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Using transcranial magnetic stimulation of the undamaged brain to identify lesion sites that predict language outcome after stroke
(2017)
Diego L. Lorca-Puls
;
GAJARDO VIDAL, ANDREA ELIZABETH
;
Mohamed L. Seghier
;
Alexander P. Leff
;
Varun Sethi
Scopus© Citations 20 1
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Erratum to: Reply: Broca’s area: why was neurosurgery neglected for so long when seeking to re-establish the scientific truth? <i>and</i> Where is the speech production area? Evidence from direct cortical electrical stimulation mapping
(2021)
Diego L Lorca-Puls
;
GAJARDO VIDAL, ANDREA ELIZABETH
;
David W Green
;
Cathy J Price
2
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Assessing processing speed and its neural correlates in the three variants of primary progressive aphasia with a non-verbal tablet-based task
(2024)
GAJARDO VIDAL, ANDREA ELIZABETH
;
Maxime Montembeault
;
Diego L. Lorca-Puls
;
Abigail E. Licata
;
Rian Bogley
Scopus© Citations 4 1
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The impact of pre-stroke formal education on language test performance in aphasic and non-aphasic stroke survivors
(Informa UK Limited, 2024-12-05)
Sophie M. Roberts
;
Rachel Bruce
;
Thomas M. H. Hope
;
Sharon Geva
;
Storm Anderson
2Scopus© Citations 3
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Dissociating the functions of three left posterior superior temporal regions that contribute to speech perception and production
(2021)
Justyna O. Ekert
;
GAJARDO VIDAL, ANDREA ELIZABETH
;
Diego L. Lorca-Puls
;
Thomas M.H. Hope
;
Fred Dick
1Scopus© Citations 15
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A functional dissociation of the left frontal regions that contribute to single word production tasks
(2021)
Justyna O. Ekert
;
Diego L. Lorca-Puls
;
GAJARDO VIDAL, ANDREA ELIZABETH
;
Jennifer T. Crinion
;
Thomas M.H. Hope
2Scopus© Citations 12
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How distributed processing produces false negatives in voxel-based lesion-deficit analyses
(2018)
GAJARDO VIDAL, ANDREA ELIZABETH
;
Diego L. Lorca-Puls
;
Jennifer T. Crinion
;
Jitrachote White
;
Mohamed L. Seghier
1Scopus© Citations 27
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Network anatomy in logopenic variant of primary progressive aphasia
(2023)
Maria Luisa Mandelli
;
Diego L. Lorca‐Puls
;
Sladjana Lukic
;
Maxime Montembeault
;
GAJARDO VIDAL, ANDREA ELIZABETH
<jats:title>Abstract</jats:title><jats:p>The logopenic variant of primary progressive aphasia (lvPPA) is a neurodegenerative syndrome characterized linguistically by gradual loss of repetition and naming skills resulting from left posterior temporal and inferior parietal atrophy. Here, we sought to identify which specific cortical loci are initially targeted by the disease (epicenters) and investigate whether atrophy spreads through predetermined networks. First, we used cross‐sectional structural MRI data from individuals with lvPPA to define putative disease epicenters using a surface‐based approach paired with an anatomically fine‐grained parcellation of the cortical surface (i.e., HCP‐MMP1.0 atlas). Second, we combined cross‐sectional functional MRI data from healthy controls and longitudinal structural MRI data from individuals with lvPPA to derive the epicenter‐seeded resting‐state networks most relevant to lvPPA symptomatology and ascertain whether functional connectivity in these networks predicts longitudinal atrophy spread in lvPPA. Our results show that two partially distinct brain networks anchored to the left anterior angular and posterior superior temporal gyri epicenters were preferentially associated with sentence repetition and naming skills in lvPPA. Critically, the strength of connectivity within these two networks in the neurologically‐intact brain significantly predicted longitudinal atrophy progression in lvPPA. Taken together, our findings indicate that atrophy progression in lvPPA, starting from inferior parietal and temporoparietal junction regions, predominantly follows at least two partially nonoverlapping pathways, which may influence the heterogeneity in clinical presentation and prognosis.</jats:p>
2Scopus© Citations 24

GAJARDO VIDAL, ANDREA ELIZABETH

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