Immune-related IncRNA LINC00944 responds to variations in ADAR1 levels and it is associated with breast cancer prognosis
Journal
Life Sciences
ISSN
0024-3205
Date Issued
2021
Author(s)
Pamela R. de Santiago
Alejandro Blanco
Fernanda Morales
Katherine Marcelain
Olivier Harismendy
Marcela Sjöberg Herrera
Type
Resource Types::text::journal::journal article
URL Institutional Repository
Cite this document
De Santiago, P. R., Blanco, A., Morales, F., Marcelain, K., Harismendy, O., Sjöberg Herrera, M., & Armisén, R. (2021). Immune-related IncRNA LINC00944 responds to variations in ADAR1 levels and it is associated with breast cancer prognosis. Life Sciences, 268, 118956. https://doi.org/10.1016/j.lfs.2020.118956
Subjects
breast cancer
;
cancer
;
lncrnas
;
linc00944
;
adar1
;
prognosis
;
tumor-infiltrating lymphocytes
;
bioinformatics
;
guilt by association analysis
;
adenosine deaminase
;
adult
;
apoptosis
;
biomarkers, tumor
;
breast neoplasms
;
cell line, tumor
;
female
;
gain of function mutation
;
gene expression regulation, neoplastic
;
gene knockdown techniques
;
humans
;
kaplan-meier estimate
;
lymphocytes, tumor-infiltrating
;
middle aged
;
prognosis
;
rna, long noncoding
;
rna-binding proteins
;
adenosine deaminase
;
adenosine deaminase acting on rna 1
;
caspase 3
;
caspase 8
;
estrogen receptor
;
long untranslated rna
;
long untranslated rna 944
;
progesterone receptor
;
protein bax
;
protein bcl 2
;
protein bcl xl
;
rna
;
tumor marker
;
unclassified drug
;
adar protein, human
;
adenosine deaminase
;
long untranslated rna
;
rna binding protein
;
tumor marker
;
adult
;
age
;
apoptosis
;
article
;
breast cancer
;
breast cancer cell line
;
cancer diagnosis
;
cancer immunology
;
cancer patient
;
cancer prognosis
;
controlled study
;
down regulation
;
female
;
gene expression level
;
gene knockdown
;
gene overexpression
;
genetic association
;
genetic variation
;
human
;
human cell
;
human tissue
;
protein expression
;
signal transduction
;
t lymphocyte
;
tumor associated leukocyte
;
tumor volume
;
upregulation
;
breast tumor
;
gain of function mutation
;
gene expression regulation
;
genetics
;
immunology
;
kaplan meier method
;
metabolism
;
middle aged
;
mortality
;
pathology
;
prognosis
;
tumor cell line