Aspirin and N‐acetylcysteine co‐administration markedly inhibit chronic ethanol intake and block relapse binge drinking: Role of neuroinflammation‐oxidative stress self‐perpetuation

Journal
Addiction Biology
ISSN
1355-6215
1369-1600
Date Issued
2019
Author(s)
Yedy Israel
María Elena Quintanilla
Paola Morales
Daniela Santapau
Mario Herrera‐Marschitz
Type
Resource Types::text::journal::journal article
Scopus ID
2-s2.0-85075459589
WoS ID
WOS:000496644300001
DOI
URL
URL Institutional Repository
Cite this document
Israel, Y., Quintanilla, M. E., Ezquer, F., Morales, P., Santapau, D., Berríos‐Cárcamo, P., Ezquer, M., Olivares, B., & Herrera‐Marschitz, M. (2021). Aspirin and N‐acetylcysteine co‐administration markedly inhibit chronic ethanol intake and block relapse binge drinking: Role of neuroinflammation‐oxidative stress self‐perpetuation. Addiction Biology, 26(1), e12853. https://doi.org/10.1111/adb.12853
Project(s)
Subjects
acetylsalicylic acid

; 

asa

; 

astrocytosis

; 

glutamate

; 

microglia

; 

oxidative stress

; 

acetylcysteine

; 

alcohol drinking

; 

alcoholism

; 

animals

; 

anti-inflammatory agents, non-steroidal

; 

aspirin

; 

binge drinking

; 

chronic disease

; 

ethanol

; 

excitatory amino acid transporter 2

; 

female

; 

oxidative stress

; 

rats

; 

recurrence

; 

self administration

; 

acetylcysteine

; 

acetylsalicylic acid

; 

alcohol

; 

calcium binding protein

; 

cystine

; 

glial fibrillary acidic protein

; 

glutamic acid

; 

ionized calcium binding adaptor molecule 1

; 

unclassified drug

; 

vesicular glutamate transporter 1

; 

acetylcysteine

; 

acetylsalicylic acid

; 

alcohol

; 

excitatory amino acid transporter 2

; 

nonsteroid antiinflammatory agent

; 

adult

; 

albumin blood level

; 

alcohol blood level

; 

alcohol consumption

; 

alcoholism

; 

amino acid transport

; 

animal cell

; 

animal experiment

; 

animal model

; 

animal tissue

; 

antiinflammatory activity

; 

antioxidant activity

; 

article

; 

astrocyte

; 

astrocytosis

; 

behavior

; 

binge drinking

; 

combination chemotherapy

; 

controlled study

; 

drug potentiation

; 

enzyme activity

; 

female

; 

hippocampal neuronal culture

; 

immunoreactivity

; 

low drug dose

; 

microglia

; 

monotherapy

; 

nervous system inflammation

; 

nonhuman

; 

oxidative stress

; 

prefrontal cortex

; 

presynaptic inhibition

; 

priority journal

; 

rat

; 

relapse

; 

self perpetuation

; 

treatment duration

; 

wistar rat

; 

alcoholism

; 

animal

; 

binge drinking

; 

chronic disease

; 

drinking behavior

; 

drug effect

; 

drug self administration

; 

oxidative stress

; 

recurrent disease