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  4. Over-activated hemichannels: A possible therapeutic target for human diseases
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Over-activated hemichannels: A possible therapeutic target for human diseases

Journal
Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
ISSN
0925-4439
Date Issued
2021
Author(s)
Mauricio A. Retamal
Facultad de Medicina Clínica Alemana Universidad del Desarrollo  
Ainoa Fernandez-Olivares
Facultad de Medicina Clínica Alemana Universidad del Desarrollo  
Jimmy Stehberg
Type
Resource Types::text::journal::journal article
Scopus ID
2-s2.0-85112451582
WoS ID
WOS:000697153700016
DOI
10.1016/j.bbadis.2021.166232
URL
https://investigadores.udd.cl/handle/123456789/6524
URL Institutional Repository
http://hdl.handle.net/11447/5113
Cite this document
Retamal, M. A., Fernandez-Olivares, A., & Stehberg, J. (2021). Over-activated hemichannels: A possible therapeutic target for human diseases. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 1867(11), 166232. https://doi.org/10.1016/j.bbadis.2021.166232
Project(s)
Carbon monoxide inhibits Cx46 HCs through a lipid peroxidation-dependent process which increases Cx46- Ca2+ sensitivity  
Subjects
connexins

; 

hemichannels

; 

channelopathies

; 

human diseases

; 

cell death

; 

animals

; 

cell line

; 

connexins

; 

drug therapy

; 

humans

; 

models, animal

; 

signal transduction

; 

antibody

; 

gap junction protein

; 

peptide

; 

small molecule transport agent

; 

gap junction protein

; 

article

; 

brain hemorrhage

; 

cell function

; 

cell survival

; 

cerebrovascular accident

; 

channelopathy

; 

chronic pain

; 

connexon

; 

epilepsy

; 

food intake

; 

heart disease

; 

human

; 

kidney disease

; 

lung injury

; 

metabolic syndrome x

; 

nonhuman

; 

protein targeting

; 

regulatory mechanism

; 

retinopathy

; 

spinal cord injury

; 

treatment outcome

; 

wound healing

; 

animal

; 

animal model

; 

cell line

; 

drug effect

; 

drug therapy

; 

metabolism

; 

procedures

; 

signal transduction
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