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  4. N-Acetylcysteine and Acetylsalicylic Acid Inhibit Alcohol Consumption by Different Mechanisms: Combined Protection
Details

N-Acetylcysteine and Acetylsalicylic Acid Inhibit Alcohol Consumption by Different Mechanisms: Combined Protection

Journal
Frontiers in Behavioral Neuroscience
ISSN
1662-5153
Date Issued
2020
Author(s)
María Elena Quintanilla
EZQUER, EDUARDO FERNANDO  
Facultad de Medicina Clínica Alemana Universidad del Desarrollo  
Paola Morales
EZQUER, EDUARDO MARCELO  
Facultad de Medicina Clínica Alemana Universidad del Desarrollo  
OLIVARES, MARIA BELEN  
Facultad de Medicina Clínica Alemana Universidad del Desarrollo  
Mario Herrera-Marschitz
Daniela Santapau
Facultad de Medicina Clínica Alemana Universidad del Desarrollo  
Yedy Israel
Facultad de Medicina Clínica Alemana Universidad del Desarrollo  
Type
Resource Types::text::journal::journal article
Scopus ID
2-s2.0-85089473010
WoS ID
WOS:000561678500001
DOI
10.3389/fnbeh.2020.00122
URL
https://investigadores.udd.cl/handle/123456789/4554
URL Institutional Repository
http://hdl.handle.net/11447/4190
Cite this document
Quintanilla, M. E., Ezquer, F., Morales, P., Ezquer, M., Olivares, B., Santapau, D., Herrera-Marschitz, M., & Israel, Y. (2020). N-acetylcysteine and acetylsalicylic acid inhibit alcohol consumption by different mechanisms: Combined protection. Frontiers in Behavioral Neuroscience, 14, 122. https://doi.org/10.3389/fnbeh.2020.00122
Project(s)
The Oxidative stress-neuroinflammation Synergy: Role in the perpetuation of alcohol intake and therapeutic avenues  
Mesenchymal stem cells-secretome provides protection in neonatal hypoxiareoxygenation. Mechanisms and translational studies  
The opioid misuse crisis; a global health concern. Possible translational avenues  
Subjects
aspirin

; 

ethanol

; 

n-acetylcysteine

; 

rats

; 

relapse

; 

2 amino 2 (2 carboxycyclopropyl) 3 (xanthen 9 yl)propionic acid

; 

acetylcysteine

; 

acetylsalicylic acid

; 

glutamic acid

; 

glutathione

; 

glutathione disulfide

; 

lipoxin a

; 

prostaglandin synthase

; 

salazosulfapyridine

; 

transcription factor nrf2

; 

adult

; 

alcohol consumption

; 

alcoholism

; 

animal experiment

; 

animal model

; 

animal tissue

; 

antiinflammatory activity

; 

antioxidant activity

; 

antioxidant responsive element

; 

article

; 

astrocyte

; 

cell activation

; 

controlled study

; 

drug acetylation

; 

drug effect

; 

drug mechanism

; 

female

; 

gene expression regulation

; 

gene overexpression

; 

hippocampus

; 

immunofluorescence

; 

microglia

; 

nervous system inflammation

; 

neuroprotection

; 

nonhuman

; 

oxidative stress

; 

rat

; 

relapse

; 

signal transduction

; 

wistar rat
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