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  4. ADAR1 Transcriptome editing promotes breast cancer progression through the regulation of cell cycle and DNA damage response
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ADAR1 Transcriptome editing promotes breast cancer progression through the regulation of cell cycle and DNA damage response

Journal
Biochimica et Biophysica Acta (BBA) - Molecular Cell Research
ISSN
0167-4889
Date Issued
2020
Author(s)
Eduardo A. Sagredo
Alfredo I. Sagredo
Alejandro Blanco
Pamela Rojas De Santiago
RIVAS VERA, SOLANGE VERÓNICA  
Facultad de Medicina Clínica Alemana Universidad del Desarrollo  
Rodrigo Assar
Paola Pérez
Katherine Marcelain
ARMISEN YAÑEZ, RICARDO AMADO  
Facultad de Medicina Clínica Alemana Universidad del Desarrollo  
Type
Resource Types::text::journal::journal article
Scopus ID
2-s2.0-85084984781
WoS ID
WOS:000536760800001
DOI
10.1016/j.bbamcr.2020.118716
URL
https://investigadores.udd.cl/handle/123456789/5797
URL Institutional Repository
http://hdl.handle.net/11447/3720
Cite this document
Sagredo, E. A., Sagredo, A. I., Blanco, A., Rojas De Santiago, P., Rivas, S., Assar, R., Pérez, P., Marcelain, K., & Armisén, R. (2020). ADAR1 Transcriptome editing promotes breast cancer progression through the regulation of cell cycle and DNA damage response. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research, 1867(8), 118716. https://doi.org/10.1016/j.bbamcr.2020.118716
Subjects
breast cancer

; 

dna damage response

; 

proliferation

; 

rna editing

; 

rna stability

; 

adenosine deaminase

; 

breast neoplasms

; 

cell cycle

; 

cell line, tumor

; 

cell proliferation

; 

disease progression

; 

dna damage

; 

female

; 

humans

; 

mcf-7 cells

; 

rna editing

; 

rna stability

; 

rna, messenger

; 

rna-binding proteins

; 

transcriptome

; 

adenosine deaminase

; 

double stranded rna

; 

messenger rna

; 

transcriptome

; 

adar1 protein, human

; 

adenosine deaminase

; 

messenger rna

; 

rna binding protein

; 

transcriptome

; 

apoptosis

; 

article

; 

breast cancer

; 

cancer growth

; 

cell cycle regulation

; 

cell proliferation

; 

cell viability

; 

clinical outcome

; 

controlled study

; 

dna damage response

; 

dna replication

; 

human

; 

human cell

; 

priority journal

; 

rna editing

; 

breast tumor

; 

cell cycle

; 

disease exacerbation

; 

dna damage

; 

female

; 

genetics

; 

mcf-7 cell line

; 

metabolism

; 

pathology

; 

physiology

; 

rna stability

; 

tumor cell line
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