IDIAQUEZ RIOS, JUAN FRANCISCO
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IDIAQUEZ RIOS, JUAN FRANCISCO
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jidiaquez@udd.cl
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57190297440

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Publication

Response to “Synucleinopathy: Treatment of Supine Hypertension”

2021 , IDIAQUEZ RIOS, JUAN FRANCISCO , IDIAQUEZ, JUAN , Juan Carlos Casar , Italo Biaggioni

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Recessive dystrophic epidermolysis bullosa results in painful small fibre neuropathy

2017 , Sofia von Bischhoffshausen , Dinka Ivulic , Paola Alvarez , Victor C. Schuffeneger , Juan Idiaquez , Constanza Fuentes , Pilar Morande , Ignacia Fuentes , FRANCIS PALISSON ETCHARREN , David L. H. Bennett , Margarita Calvo

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Publication

Cardiovascular responses to isometric handgrip exercise in young patients with recurrent vasovagal syncope

2018 , Juan Idiaquez , Juan Francisco Idiaquez , Rodrigo Iturriaga

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Evaluación clínica de las Disautonomías

2020 , Juan Idiáquez C , IDIAQUEZ RIOS, JUAN FRANCISCO , Eduardo Benarroch

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Publication

Epidemiology of chronic inflammatory demyelinating polyneuropathy in the South-Eastern area of Santiago, Chile

2020 , Gabriel Cea , IDIAQUEZ RIOS, JUAN FRANCISCO , Rodrigo Salinas , MATAMALA CAPPONI, JOSE MANUEL , Roque Villagra , Andrés Stuardo

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Assessing diabetic polyneuropathy in Spanish‐speaking patients: Translation and validation of the Toronto Clinical Neuropathy Score

2023 , IDIAQUEZ RIOS, JUAN FRANCISCO , Ignacio Acosta , Alberto Prat , Francesca Gattini , Francisca Pino , Carolina Barnett‐Tapia

AbstractBackground and AimsDiabetic sensorimotor polyneuropathy (DSP) is a common complication of diabetes. The Toronto Clinical Neuropathy Score (TCNS) is a useful tool for detecting DSP. However, it is not available in Spanish. The study aimed to translate and culturally adapt the TCNS and modified (mTCNS) scales into Spanish and evaluate their measurement properties.MethodsA multistep forward‐backward method was used for translation and cultural adaptation. A panel of physicians subjected the final Spanish versions of TCNS and mTCNS (TCÑS, mTCÑS) to cognitive debriefing. Consecutive patients with diabetes mellitus and DSP were recruited from an outpatient clinic, and the TCÑS and mTCÑS were tested for construct validity, along with other measures.ResultsThe internal consistency of both TCÑS and mTCÑS was excellent, as evidenced by Cronbach's Alpha coefficients of 0.83 and 0.85, respectively. Furthermore, there was a robust positive correlation between TCÑS and mTCÑS. In addition, TCÑS was found to exhibit a strong negative correlation with sural sensory nerve action potential amplitude (r = −0.9206) and peroneal compound motor action potential amplitude (r = −0.729), while demonstrating a positive and strong correlation with the Michigan Neuropathy Screening Instrument (r = 0.713).InterpretationThe TCÑS and mTCÑS are reliable and valid translations of the original TCNS. The TCÑS and mTCÑS can be used to diagnose and measure the severity of neuropathy in Spanish‐speaking patients with diabetes.

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Adhesión al tratamiento farmacológico y descripción de sus factores asociados en pacientes con miastenia grave

2018 , IDIAQUEZ RIOS, JUAN FRANCISCO , Sofía González Too- Kong , Jorge Lasso Peñafiel , Carolina Barnett

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Clinical and Electrophysiological Features of Chronic Inflammatory Demyelinating Polyneuropathy in Chile.

2019 , IDIAQUEZ RIOS, JUAN FRANCISCO , Gabriel Cea , José Manuel Matamala , Rodrigo Salinas , Roque Villagra , Andrés Stuardo

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Neurogenic Orthostatic Hypotension. Lessons From Synucleinopathies

2020 , IDIAQUEZ RIOS, JUAN FRANCISCO , IDIAQUEZ RIOS, JUAN FRANCISCO , Juan Carlos Casar , Italo Biaggioni

Abstract Maintenance of upright blood pressure critically depends on the autonomic nervous system and its failure leads to neurogenic orthostatic hypotension (NOH). The most severe cases are seen in neurodegenerative disorders caused by abnormal α-synuclein deposits: multiple system atrophy (MSA), Parkinson’s disease, Lewy body dementia, and pure autonomic failure (PAF). The development of novel treatments for NOH derives from research in these disorders. We provide a brief review of their underlying pathophysiology relevant to understand the rationale behind treatment options for NOH. The goal of treatment is not to normalize blood pressure but rather to improve quality of life and prevent syncope and falls by reducing symptoms of cerebral hypoperfusion. Patients not able to recognize NOH symptoms are at a higher risk for falls. The first step in the management of NOH is to educate patients on how to avoid high-risk situations and providers to identify medications that trigger or worsen NOH. Conservative countermeasures, including diet and compression garments, should always precede pharmacologic therapies. Volume expanders (fludrocortisone and desmopressin) should be used with caution. Drugs that enhance residual sympathetic tone (pyridostigmine and atomoxetine) are more effective in patients with mild disease and in MSA patients with spared postganglionic fibers. Norepinephrine replacement therapy (midodrine and droxidopa) is more effective in patients with neurodegeneration of peripheral noradrenergic fibers like PAF. NOH is often associated with other cardiovascular diseases, most notably supine hypertension, and treatment should be adapted to their presence.

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Publication

Electrophysiological severity of carpal tunnel syndrome according to age in adult patients

2017 , Pilar Vicuña , Juan Francisco Idiáquez , Paula Jara , Francisca Pino , Marcela Cárcamo , Gabriel Cavada , Renato Verdugo

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